Susan Marsh
Education
PhD in human movement studies, The University of Queensland, Brisbane, Australia
Bachelor of Science (Honours I), The University of Queensland, Brisbane, Australia
Bachelor of Health Science, Griffith University, Gold Coast, Australia
Bachelor of Education (Secondary), Queensland University of Technology, Brisbane, Australia
Additional Training
Postdoctoral Scholar, Division of Cardiovascular Disease, University of Alabama at Birmingham, Birmingham, AL
Fundamentals of Online Teaching Certificate, University of Wisconsin-Madison
Teaching
PharDSci 512 Integrated Pharmacology II
PharDSci 532 Integrated Pharmacology III
Pharmacy 572 Physical Activity Prescription in the Pharmacy
Research Interests
Roles of exercise and protein O-GlcNAcylation in cardiac hypertrophy
Interactions between exercise and commonly used cardiovascular medications
Selected Publications
Johnson, EJ., Dieter, BP., Marsh, SA. Establishing strategic exercise prescriptions for cardiac hypertrophy: Evidence for distinct effects of exercise in different hypertrophic disorders. Life Sciences. 123: 100–106, 2015. [PMID: 25632833]
Medford, HM., Marsh, SA. The role of O-GlcNAc transferase (OGT) in regulating gene transcription of developing and failing hearts. Future Cardiology. 10(6): 801-812, 2014. [PMID: 25495821]
Marsh, SA., Collins, HE., Chatham, JC. Protein O-GlcNAcylation and Cardiovascular (Patho)physiology. Journal of Biological Chemistry. 89(50): 34449–34456, 2014. [PMID: 25336635]
Rocco, AB., LeValley, JC., Eldridge, JA., Marsh, SA., Rodgers, BD. A novel protocol for assessing exercise performance and dystropathophysiology in the mdx mouse. Muscle & Nerve. 50(4): 541-548, 2014. [PMID: 24449511]
Medford, HM., Cox, EJ., Miller, LE., Marsh, SA. Consuming a Western diet for two weeks suppresses fetal genes in mouse hearts. American Journal of Physiology – Regulatory, Integrative and Comparative Physiology. 306(8): R519-26, 2014. [PMID: 24523346]
Cox, EJ., Marsh, SA. A systematic review of fetal genes as biomarkers of cardiac hypertrophy in rodent models of diabetes. PLOS ONE. 9(3): e92903, 2014. [PMID: 24663494]
Cox, EJ., Marsh, SA. Exercise and diabetes have opposite effects on the assembly and O-GlcNAc modification of the mSin3A/HDAC1/2 complex in the heart. Cardiovascular Diabetology. 12: 101, 2013. [PMID: 23835259; PMCID: PMC3708830]
Medford, HM., Porter, K., Marsh, SA. Immediate effects of a single exercise bout on protein O-GlcNAcylation and chromatin regulation of cardiac hypertrophy. American Journal of Physiology – Heart and Circulatory Physiology. 305(1): H114-123, 2013. [PMID: 23624624]
Medford, HM., Chatham, JC., Marsh, SA. Chronic ingestion of a western diet increases O-linked-β-N-acetylglucosamine (O-GlcNAc) protein modification in the heart. Life Sciences. 90(23-24): 883-8, 2012. [PMID: 22575823]
Porter, K., Medford, HM., McIntosh, CM., Marsh, SA. Cardioprotection requires flipping the ‘posttranslational modification’ switch. Life Sciences. 90(3-4): 89-98, 2012. [PMID: 22154907]